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Severe ADAMTS13 Deficiency 

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Intracellular Pathways

medical illustration of ADAMTS13 is an enzyme produced by the vascular endothelium that cleaves von Willebrand Factor, a protein that mediates platelet adhesion and aggregation at sites of vascular injury. Deficient levels of ADAMTS13 allows platelets to bind to uncleaved von Willebrand Factor multimers and leads to thrombotic microangiopathy.
Next medical illustration of Destruction of virulent microbe strains is hampered by the microbe’s protective outer membrane. Stronger antimicrobial drugs are able to penetrate and destroy the outer membrane and expose the more vulnerable inner membrane.

Microbe Destruction

ADAMTS13 is an enzyme produced by the vascular endothelium that cleaves von Willebrand Factor, a protein that mediates platelet adhesion and aggregation at sites of vascular injury. Deficient levels of ADAMTS13 allows platelets to bind to uncleaved von Willebrand Factor multimers and leads to thrombotic microangiopathy.

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© Edmond Alexander