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Cyclooxygenases and NSAIDS 

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Diabetic Glomerulus

medical illustration of COX-2 selective NSAIDs were developed to maintain analgesia efficacy while minimizing the side effects associated with COX-1 inhibition. The cyclooxygenase enzyme is anchored to the endoplasmic reticulum membrane. Above it, released prostaglandin hormones soar into the cell. The COX-2 selective NSAID is blocked from the channel of access for COX-1, but gains entry at COX-2 site, and thereby inhibits the active site for pain prostaglandin creation.
Next medical illustration of Tivantinib™ is designed to block the activity of c-MET, a molecule that plays multiple key roles in human cancer including cancer cell growth,survival, angiogenesis, invasion and metastasis in nsNSCLC (non squamous Non-Small Cell Lung Cancer).

Tivantinib™ Mode of Action in nsNSCLC

COX-2 selective NSAIDs were developed to maintain analgesia efficacy while minimizing the side effects associated with COX-1 inhibition. The cyclooxygenase enzyme is anchored to the endoplasmic reticulum membrane. Above it, released prostaglandin hormones soar into the cell. The COX-2 selective NSAID is blocked from the channel of access for COX-1, but gains entry at COX-2 site, and thereby inhibits the active site for pain prostaglandin creation.

Keywords: Airbrush, Color, 3D, Editorial, Cell biology / Histology, Disease Management, Molecular Biology, Pharmacology, Mechanism of Action (MOA)

© Cynthia Turner